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Importantly, these objective biomarkers of AA intakes significantly predict T2D risk, corroborating our findings based on long-term self-reported AA intake

Although these species are generated during normal cellular metabolism and participate in essential physiological processes like signal transduction and gene expression at low concentrations (Krishnamurthy et al., 2024), their excessive accumulation or insufficient scavenging leads to oxidative stress, causing damage to vital biomolecules and cellular structures

4) and a form of cell death exhibiting ferroptosis-like features in thyroid cancer cells [142]