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P53 impairs the cystine/glutamate antiporter by silencing SLC7A11, its key component, resulting in cellular cysteine depletion and triggering ferroptosis

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Boldfaced data indicate that the differences were statistically significant (Student's test 10 days) and not in planktonic cultures

Glycolysis and glutaminolysis enhancement are two of the most common but vital modalities in TME ( In this review, we discuss the metabolic reprogramming in MM, which is associated with a hypoxic, acidic, and nutritionally deficient TME, and how these changes impede the function of immune cells