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In hepatocellular carcinoma, AMPK activation by ferroptosis inducers like erastin, sorafenib, and sulfasalazine inhibits branched chain amino acid transaminase 2 (BCAT2) transcription, modulating glutamate levels and preventing ferroptosis [69]

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Taking account of the mechanisms of ferroptosis, GSH-depleted methods have been extensively exploited for antitumor therapy based on GSH-enhanced GPX4 inactivity and LPO accumulation

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