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Alterations in other melanogenesis-related genes, such as tyrosinase-related proteins (TRPs) and melanocyte-inducing transcription factor (MITF), further contribute to graying

Socs1: phosphorylation, dimerization and tumor suppression

Concurrently, its anti-inflammatory effects are mediated by inhibiting nuclear factor-kappa B (NF-B), a protein complex that controls transcription of DNA, cytokine production, and cell survival

Increased amyloid beta precursor protein (APP) and beta-secretase 1 (BACE1) transcription was the result of this histone hyperacetylation and DNA hypomethylation, potentially as a result of an increase in NF-B activity (Gu et al., 2013)