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Dysfunction in key components of this network leads to the disruption of Cu homeostasis, resulting in fatal disorders such as Wilson disease, which is caused by mutations in the hepatic Cu efflux transporter ATP7B

From its purported role in extracellular matrix modulation, gene expression resetting, anti-inflammatory/antioxidant signaling, to tissue repair mechanisms, the peptide provides a unique convergence of metal-ion chemistry, peptide biology, and regenerative mechanisms

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