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Luebke et al., 2014)

wrote the final manuscript, which all authors critically reviewed for intellectual content and approved

Based on these findings, the reversible and short-lasting inhibition of MAO-B by safinamide administration, the absence of MAO inhibition during LJP-1207 treatment or the lack of a propargylamine moiety in both molecules, which have intrinsic neuroprotective activity [109, 110], could contribute to their ineffectiveness in our AGIHL model