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Studies in SLC25A19 knockout mice and in carriers of the SLC25A19 mutation, responsible for the Amish lethal microcephaly (MCPHA, see below) show, respectively, virtually undetectable and markedly reduced mitochondrial TPP levels in mouse embryonic fibroblasts and human lymphoblasts (56), which were completely restored upon addition of TPP to the PDH and KGDH complex assay mixtures (56)

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CARM1 contributes to skeletal muscle wasting by mediating FoxO3 activity and promoting myofiber autophagy

PMID: 10373343 Reported Bone mineral content (DXA, rat) Increased bone mineral content via bone-dimension expansion (young adult female rat) Svensson et al