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No significant side effects, or exacerbation of vitiligo were observed during or after treatment with NCTF 135 in the following 6 months (five patients) and 12 months (two patients)

In contrast to melanomas, which acquire additional driver mutations, nevi usually enter a suppressive state of replicative senescence which is regulated by the tumor suppressor gene CDKN2A via its proteins, p14 and p16, and various transcriptional controls of the cell cycle (30, 31)

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