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However, factors such as chronic nutrient excess, physical inactivity, genetic predisposition, and aging can drive AT dysfunction, overwhelming its adaptive capacity ( Multiple mechanisms underlie AT dysfunction, including mitochondrial impairment, ER stress, inflammatory pathway activation, and extracellular matrix (ECM) remodeling

Synthesis and study of L-dopa-glutathione codrugs as new anti-Parkinson agents with free radical scavenging properties

The effect of preoperative melatonin on nuclear erythroid 2-related factor 2 activation in patients undergoing coronary artery bypass grafting surgery

Rab 1a, 1b, Rab3 (a, b, c, d), Rab5a, Rab7a Rab7L1, 8a, 8b, 10, 13, 15, 18, 19, 27a, 38, 40a and 42 (Gitler and Tsuiji, 2016