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3.5 Immune-oriented nanotherapeutics: systemic priming and intratumoral reprogramming Immune-oriented nanotherapeutics alter the balance between antitumor effector mechanisms and suppressive immune networks

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A study proved that p53 could bind to dipeptidyl-peptidase 4 (DPP4) to reduce the DPP4-dependent oxidization of PUFAs to inhibit ferroptosis in colorectal cancer cells[46]