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Increasing the dose beyond the effective range adds little benefit regardless of body weight

Trehalose, a novel mTOR-independent autophagy enhancer, accelerates the clearance of mutant huntingtin and -synuclein

Next, we will further validate the roles of YAP and TAZ in the disease and confirm the effectiveness of therapies that inhibit these proteins, offering patients better outcomes." In addition to pulmonary fibrosis, YAP and TAZ are also implicated in heart, liver and kidney fibrosis, suggesting that therapies targeting these proteins could offer broader therapeutic potential for a range of fibrotic diseases