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De Michele, S

11 Disturbances in iron homeostasis in liver diseases may be a manifestation of: Active inflammation Increase in liver iron Parenchymal damage with increased iron release from damaged/dead hepatocytes Acute: Acute hepatitis: Serum ferritin concentrations correlated with serum alanine transferase activity, especially in those with viral hepatitis

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Notably, when administered at a dose of 62.5 mg/kg, NMN produced optimal treatment effects compared to other doses (500, 250, 125, and 31.25 mg/kg), suggesting that high dosage of NMN may introduce adverse effects on post-ischemic neurons