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Description

Conditioned medium from Se/LDH@PEG-M-treated macrophages attenuates chondrocyte apoptosis, inflammation and catabolism compared to LPS group

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A: LASSS neutralizes reactive molecules to prevent nitration and appears to induce a subtle structural shift in HGF itself

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Our results show blunted angiogenic signaling despite rapid and sustained HIF-1 stabilization in seal cells

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