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In this review, we highlight the emerging evidence linking amino acid metabolic pathwaysparticularly those involving arginine, proline, glycine, and serineto the initiation and progression of pulmonary fibrosis

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However, different pattern of elemental abnormalities found between male and female rats, normal liver Fe level despite of diminished content of the element in ketogenic fodder, the lack of abnormalities in the range of low-Z elements for female rats, suggest that the diminished level of measured elements in the fodder is not the only reason of the found liver anomalies