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Three mutations in the electron transport chains complex I (NADH dehydrogenase) account for 80% of patients with Lebers Hereditary Optic Neuropathy (Schrier and Falk, 2011)

Increasing evidence supports a role for altered energy metabolism in the resistance of tumours to anti-cancer therapy, suggesting that targeting tumour metabolism may have potential as a novel therapeutic strategy to boost treatment response

The optimum number of clusters ( K ) was determined using the cross-validation errors provided by ADMIXTURE ver

In contrast, the amlodipine-treated group had near-normal phosphorylation levels at all three sites (Figure 6B), suggesting that the amlodipine treatment normalizes pathological metabolic signaling in cardiac tissue