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Furthermore, CRIP1 WT and R16F suppressed the interaction between p38 and MKK3 compared to the R16K mutant (Fig

Conclusions In the pathological mechanism of AML, the interaction and coordination between autophagy and metabolism play a vital role in maintaining intracellular physiological homeostasis, which is potentially a significant factor behind the resistance exhibited by AML cells during chemotherapy

DOI: 10.1007/s00109-016-1488-y PMID: 27847966 View Source Scientific Reports (Nature) 2020 Modulatory effects of BPC 157 on vasomotor tone and the Src-Cav-1-eNOS signaling pathway in rats Hsieh MJ, Lee CH, Chueh HY et al

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