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Because studies have shown that more than 60 % of RLS patients have a family history of the disease [18, 19], there has been an assumed genetic component of this disease and several genome-wide associated studies have found an increased risk for developing RLS and PLM with a few genomic regions (BTBD9, MEIS1, MAP2K5/LBXCOR1, and PTPRD) [20]

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