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SIRT1 activation by curcumin pretreatment attenuates mitochondrial oxidative damage induced by myocardial ischemia reperfusion injury

This toxic metal will further amplify lipid peroxidation through the Fenton reaction, eventually contributing to ferroptosis [203, 204]

Nonetheless, due to biological and physiological differences between a small animal model and humans, such as a thinner cartilage, possible spontaneous healing of defects, biomechanics, and physical loading in small animals, thus, an additional chondral defect repair study using minipigs should be conducted to confirm these effects

Interestingly, an in-depth analysis via LCMS/MS confirmed thiol peroxidase HpTpx (18 kDa, O25151) and revealed another crucial stress-response protein, chaperonin HpGroEL (60 kDa, P42383), as significantly outcompeted targets matching the gel-based results (Fig