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Mitochondrial dysfunction-evoked DHODH acetylation is involved in renal cell ferroptosis during cisplatin-induced acute kidney injury

Ten years ago, ferroptosis was first documented as a program cell death of the body which is involved in variety of biological processes, including muscle atrophy, neuron loss, tumor growth, ischemia reperfusion and immune escape

More recently, two randomized controlled trials of DFP in amyloid-confirmed early AD produced paradoxical outcomes: patients receiving DFP deteriorated faster than those on placebo over 12 months (Lannfelt et al., 2008)

Moreover, PER2 has been reported to form a trimeric complex with p53 and the oncogenic mouse double minute-2 homolog protein (MDM2), the p53s negative regulator, thereby increasing p53 stability by blocking Mdm2-dependent ubiquitination and transcription of p53 target genes 36