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However, it has been shown that TET-mediated DNA oxidation induced by vitamin C can create a synthetic lethality, where AML cells being forced to undergo active DNA demethylation renders them hypersensitive to PARP inhibition ( Conclusion Given the numerous roles for vitamin C in biological processes that maintain and influence genomic stability, it is no surprise that vitamin C continues to be explored for potential anticancer activity

Research has demonstrated that AGEs can trigger cellular senescence via several mechanisms, including the initiation of p21 in an oxidative stress-dependent manner, upregulation of p16 expression, inhibition of autophagy by diminishing PINK1/Parkin activity, and the promotion of inflammation in renal cells (Liu et al., 2014

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There simply isnt a standard test for this condition, and any results can be somewhat inaccurate