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Additionally, genetic variations in vitamin C transporters may play a role

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Intriguingly, the replication track lengths were quite similar in T315I-Bcr-Abl-32D and 32D cells

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Mutations in GBA1 represent one of the most common genetic risk factors for PD, leading to significantly reduced GCase protein activity in cholinergic neurons and impairing -syn metabolism, thereby elevating -synuclein levels in these neurons [179]

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