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Introduction The ability to manage body oxygen stores in the face of environmental hypoxia constrains the life history of many vertebrate taxa

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Research on the role of macrophages in myocarditis has focused primarily on the M1 and M2 polarization phenotypes, with a general consensus that M1 macrophages exacerbate myocarditis, whereas M2 macrophages alleviate myocardial inflammation

While neither agent is specific for ferroptosis, these pre-clinical model studies suggest that a role for ferroptosis in the pathophysiology of IBD is worthy of greater consideration [37]