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In such conditions, insufficient peroxisomal fatty acid processing could increase mitochondrial substrate pressure, whereas persistent mitochondrial dysfunction would in turn impair completion of oxidative disposal, establishing a self-reinforcing loop of NEFA accumulation, ROS production, and necroinflammation (60, 61)

This is the case that in our current study, we clearly detected a significant increase of Fe 2+ signal (Figure 1), and noticeable changes in the balance of GPx4 and FACL4 following CSD (Figure 2)

Perilipin expression alone has been shown to be sufficient to confer PKA-mediated lipolysis in CHO cells that lack HSL (126)

L.SaezL.BlauJ.RothenfluhA.WesleyC