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While there is currently no conclusive evidence for disease-modifying efficacy on any of the numerous efforts, we summarize recent late-stage clinical trial evidence focusing on glucagon-like peptide 1 (GLP1) agonists, approaches targeting the glucocerebrosidase (GBA) pathway and inhibition of the leucinerich repeat kinase 2 (LRRK2), as well as -synuclein based treatments

Multi-methylation To further complicate the landscape of histone methylation, residues can be methylated to different extents