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In autoimmune diseases such as systemic lupus erythematosus (SLE), mitochondrial damage and the release of mitochondrial DNA (mtDNA) into the cytoplasm and extracellular space have been observed [130]

The observed delay of probe reduction by GSSG could be due to the time endogenous glutathione reductase needs to reduce the imported GSSG (Figure 1D)

*Correspondence: Tim Rolph, [email protected] This article was submitted to Obesity, a section of the journal Frontiers in Endocrinology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

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