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The physiological reserves available for detoxifying cyanide are therefore limited (Schulz et al., 1979b)

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When the body is in a Se-deficient state, the sensitivity of cells to ferroptosis increases, the antioxidant function of GPX4 and other selenoproteins against lipid peroxidation decreases, and the level of intracellular peroxidation products increases, causing cell damage and even ferroptosis, which is a landmark event of ferroptosis

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