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Some disorders of intracellular cobalamin metabolism (cblC, cblD-MMA/Hcy, cblF, and cblJ) do not only compromise the synthesis of methylcobalamin, but also the synthesis of adenosylcobalamin, cofactor of the enzyme methylmalonyl-CoA mutase, leading to elevated concentrations of both tHcy and methylmalonic acid (MMA)

It plays a crucial role in numerous bodily functions including DNA formation, red blood cell formation, and nerve maintenance

Unweighted UniFrac showed a clear separation between the microbiota of nave treatment groups and inf_CNCbl40 group, whereas some mice from the inf_CON group remained similar to the nave treatment groups (Fig