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doi: 10.1007/s00018-010-0460-1

To clarify the causal relationships and stagedependent roles of these factors, we propose a conceptual framework for AD progression (1):Early AD (causal events): Disruption of cerebral iron homeostasis leads to regional iron accumulation, which primes neurons for ferroptosis through elevated ROS and impaired antioxidant defenses (2).Transitional AD (facilitative events): Ferroptotic stress promotes A aggregation and tau hyperphosphorylation

Model fit were assessed by examining Nagelkerke R 2 of carnitine associated with fracture (0.796), which indicates a good model fit