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Supraphysiological testosterone concentrations have been shown to reduce endothelial nitric oxide synthase expression and nitric oxide bioavailability, thereby favouring vasoconstriction and increased arterial stiffness ( Oxidative stress appears to represent a central mechanistic pathway linking molecular signalling alterations with structural cardiac pathology

Therefore, redox regulation is involved in the occurrence and progression of various diseases at multiple levels and is expected to become a key entry point for therapy