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Antibodies to immune checkpoint molecules on effector T cells such as programmed cell death-1 (PD1) and cytotoxic lymphocyte antigen-4 (CTLA-4) restores the effector T cell activity which is suppressed by ligands highly expressed on the cancer cells leading consequently to not only cancer cell death, but also death of regulatory T cells in the tumor environment

The most compelling research endpoints involve mortality and disease incidence tracked over years, as demonstrated in the long-term follow-up studies showing reduced mortality in treated groups

Its mechanism and receptor specificity make it a valuable tool for researchers studying hormone rhythm dynamics, peptide-receptor interactions, and multi-pathway endocrine signaling

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