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Inflammasomes, particularly the NLRP3 inflammasome, play a central role in regulating this phase by sensing both pathogen-associated molecular patterns (PAMPs) and damage-associated molecular patterns (DAMPs) released during injury, such as those generated in thermal burns

This is the central reason the GLOW protocol works: it supports multiple bottlenecks simultaneously

Compared to literature on oxidative stress-induced microglial activation and neuroinflammatory responses, these findings emphasize the potential bridging role of exosomes in this process, deepening understanding of the mechanisms underlying HACE and its cognitive dysfunction [72]

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