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According to research, inhibiting the activities of key carbohydrate hydrolases such as intestinal -glucosidase and pancreatic -amylase could delay the degradation of polysaccharides, oligosaccharides, and disaccharides in the gastrointestinal tract, thus preventing hyperglycemia ( -glucosidase and -amylase in a dose-dependent manner ( in vivo experiment involving KKAy mice revealed that LBP significantly inhibited -glucosidase activity in the intestinal tract at 30 and 120 min after a meal, resulting in lower PBG levels at 30,90, and 120 min during OGTT ( -glucosidase and -amylase inhibitor that might antagonize glucose digestion and absorption, thus regulating the PBG peak following carbohydrate intake

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