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This leads to increased CD38 mRNA transcription, which in turn favors surface CD38 expression and, consequently, improved binding and lytic potential of the antibody ( 2.1.1.6 Other potential modulators of CD38 surface expression Surface levels of CD38 rapidly decline following in vivo treatment with mAbs, displaying prolonged recovery times that contradict the expected rapid re-synthesis of the molecule or mobilization from internal stores ( + effectors

However, homeostasis (i.e., in the stomach as stomach cytoprotection/adaptative cytoprotection, and in the other organs as organoprotection) still warrants eventual realization in more advanced practice (providing that prostaglandins analogues have no more than marginal application in gastroduodenal lesions therapy)