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EJ-1 human bladder cancer cells treated with dimethylarsinous acid (DMA(III)) or dimethylmonothioarsinic acid (DMMTA(V)) presented a 60% decrease in glutathione (GSH), indicating the involvement of oxidative stress in cell death

It is usually a matter of tissue behavior

The link between epithelial permeability and pain likely reflects the abovementioned redundancy of the system involving the gut microbiota overstimulating the immune system through a leaky IEB, leading to the release of immune mediators, such as histamine, tryptase, serotonin, polyunsaturated fatty acids (12-hydroperoxyeicosatetraenoic acid, 15-hydroxyeicosatetraenoic acid, 5-hydroxyeicosatetraenoic acid, 5-oxoeicosatetraenoic acid, and leukotriene B4) (235, 236) known to evoke sensory afferent over-stimulation and pain (230)

Below is a framework comparing safety considerations across common administration scenarios to help inform discussions with your healthcare provider