hepatic glutathione concentrations system enhancement for cardiac protection: pharmacological options against oxidative stress and ferroptosis Glutathione in liver diseases and

DGBP treatment alters protein expression and membrane association of RhoA, Rac and Rap1 but not Cdc42 We examined the effect of DGBP on levels and membrane localization of a panel of geranylgeranylated proteins including RhoA, total Rac, Cdc42 and Rap1 (Figures 3a and b)

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6), suggesting that glucose-driven oxPPP becomes the dominant route for cellular NADPH production during oxidative burst even though it may not be the dominant source at baseline, which is consistent with the large increase in oxPPP flux measured by 13 C labelling