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Although tissue-specific differences in hepcidin sensitivity, along with variations in critical thresholds, time kinetics and the compensatory capability of cellular antioxidant defense systems, may modulate iron accumulation and ferroptotic outcomes, existing evidence supports a pro-ferroptotic role of hepcidin in Kupffer cells, neurons and hepatocytes (65-67)

Any discussion of specific peptides or potential research findings is informational only and must not be interpreted as guidance for use in or on humans or animals

'No data' does not mean 'proven safe,' but glutathione lacks the mechanisms that make most supplement-drug interactions concerning and that distinction matters