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Therefore, GLPs restore cell viability and alleviate pancreatic damage through anti-apoptosis combined with other mechanisms such as anti-oxidation and management of glucose and lipid metabolic processes, reverse hyperglycemia and its associated organ damage, fundamentally delay the progression of diabetes, and exert a three-dimensional protective impact on preventing and treating diabetes and its complications (Li et al., 2019b

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Dysregulation of iron homeostasis mediated by FTH increases ferroptosis sensitivity in TP53-mutant glioblastoma

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