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Stress adaptation in a pathogenic fungus

Furthermore, additional evaluation at T2-star-weighted MRI would have accurately assessed hemosiderin deposition

Activation of ATM by oxidative stress or genotoxic damage was recently reported to activate AMPK and the tuberous sclerosis complex 2 (TSC2), which in turn participates in energy sensing and growth factor signaling to repress the kinase mTOR in the mTORC1 complex [83]

When we applied this assay to yeast cells expressing Grx1roGFP2 we found that during recovery the maximum at 405 nm decreased in intensity while the maximum at 488 nm increased (Figure 1E)