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Neurons undergoing ferroptosis release damage-associated molecular patterns (DAMPs) such as HMGB1, ATP, and lipid peroxidation products, which activate microglia and drive them toward a pro-inflammatory phenotype

CX3CR1 deficient mice could not resolve intestinal inflammation in azoxymethane (AOM)/DSS-induced colitis-associated cancer and have higher colitis score and polyps, compared with wildtype mice