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RT activates the cGAS-STING pathway in tumor cells, and while dsDNA and cGAMP can be transmitted to myeloid cells through multiple mechanisms to activate their cGAS-STING pathway, this activation is often transient and insufficient

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The Trp161Phe variant has historically been puzzling as it profoundly affects PCET catalysis without directly removing a hydrogen bond donor or acceptor 26,48,51