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The resulting Ca + overload disturbs ion homeostasis and induces mitochondrial dysfunction, oxidative stress, and neuronal edema, ultimately culminating in cell death ( Studies have shown that RIPreC significantly reduces glutamate levels in both the ischemic core and penumbra, mainly through the enhanced efflux of glutamate into peripheral blood

Post hoc analysis did show a trend toward prolonged time to non-self-sufficiency but was not statistically significant

Funding This review was supported by Grants from the National Natural Science Foundation of China (82101342 to LQ), Science and Technology Innovation Program of Hunan Province (2021SK53501 to HZ), Horizontal research Project (02220141 to HZ), and Scientific Research Launch Project for new employees of the Second Xiangya Hospital of Central South University

Prior studies have reported SalA-mediated cardioprotection, hepatoprotection, and nephroprotection through mitigation of ROS and inflammatory burden (Youssef et al