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Animal studies have revealed that mild to moderate acoustic overexposure, even when causing only a temporary threshold shift (TTS), can immediately and permanently induce cochlear synaptopathycharacterized by the loss of synaptic connections between cochlear hair cells and auditory nerve fibers, without any loss of the hair cells themselves 16,17,18

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Neither is "better" in a research sense, because the two are studied for different mechanistic questions

4e), which is regulated by PPAR and catalyses the rate-limiting step of FAO by converting acyl-CoAs into acylcarnitines allowing their subsequent mitochondrial -oxidation 42