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This review explores melatonin-mediated protective mechanisms in the retina, a likely mechanistic basis for the already published findings showing that melatonin use by humans is associated with delayed AMD, and potential clinical applications

Modifications can be made to the termini of existing CPPs or the peptide backbone can be adjusted, such as through peptide cyclization, to improve stability and cell permeability

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It follows that the T-MNS (ICG) had obvious advantages in drug accumulation and retention for CNV in vivo, as well as good penetrability in corneal tissues and a targeting effect toward blood vessels