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UDCA, a hydrophilic bile salt, enhances the expression and nuclear translocation of Nrf2 thereby increasing expression of GSH synthesizing enzymes and the GSH level through PI3K/Akt pathway [87, 4]

However, a discovery that bacterial pore-forming toxins can induce necroptosis (a programmed necrosis) also in red blood cells (RBCs) is striking and could eventually lead to a re-conception of erythrocytes as entities between life and death [25]