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The Initial Vascular Access Type Contributes to Inflammation in Incident Hemodialysis Patients

Genotyping was assessed using two similar arrays, i.e., the UK BiLEVE array and UK Biobank Axiom array

The mutations comprise of 57 amino acid substitutions (65.5%), one complete gene deletion (3, 306), one complete exon 1 deletion (16), one substitution at stop codon 255 resulting in a Serine residue (336), ten small deletions resulting in either a premature stop codon or in an in-frame amino acid residue deletion, four small insertions (335), nine nonsense mutations and four splice site mutations, resulting in aberrant splicing [Figure 12]

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