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Mathematical and evolutionary models suggest that resistant subclones often carry a fitness cost in the absence of drug, meaning that drug holidays or reduced-intensity intervals can allow sensitive populations to re-expand and suppress resistant lineages, if they compete for the same niche

However, whether the exogenous serine or glycine supports the proliferation of cancer cells is still highly debatable

DOX (Doxorubicin), CP (Cyclophosphamide), PARP-1 (Poly (ADP-Ribose) Polymerase 1), Bcl-2 (B-Cell Lymphoma 2), AC (Anticancer Chemotherapy), IL-3 (Interleukin-3), NF-B (Nuclear Factor Kappa-Light-Chain-Enhancer of Activated B Cells), COX-2 (Cyclooxygenase-2), IL-3 (Interleukin-3), CYP (Cyclophosphamide), 5-FU (5-Fluorouracil), MTX (Methotrexate), SOD (Superoxide Dismutase), CAT (Catalase), GPx (Glutathione Peroxidase), GST (Glutathione S-Transferase), MDA (Malondialdehyde), ROS (Reactive Oxygen Species)