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doi:10.2174/138161210790883615 Vukojevi J, Sikiric P, Drmic D, et al

Riboflavin is essential for homocysteine metabolism, acting as a cofactor for enzymes like methylenetetrahydrofolate reductase (MTHFR) and methionine synthase reductase (MTRR)

However, restoring SubP-mediated signaling through tachykinin agonists seems like an ineffective strategy in CF because evidence suggests that the defect in SubP-mediated glandular secretion in CF is due to loss of CFTR [100]