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We hypothesize that the selenium-iodine-induced oxidative eustress acts as a signal that shunts metabolic precursors away from the phenylpropanoid pathway (which produces eugenol) and toward the methylerythritol phosphate pathway, which leads to linalool synthesis

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Recently, an increasing number of studies have demonstrated the important regulatory role played by epigenetic mechanisms, especially DNA, RNA, and protein methylation, in ferroptosis

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