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Our findings, together with previous studies identifying SHIN1 as a negative modulator of psoriasiform reactions in IMQ mouse model [15], provide a strong rationale for the development of SHIN1-based topical formulations and other metabolism-targeted therapeutic strategies for psoriasis and potentially for other IL-22-driven hyperproliferative skin disorders, including NMSC [52]

Optical, morphological and biological analysis of zinc oxide nanoparticles using Papaver somniferum

Local arterial administration of acidified malonate as an adjunct therapy to mechanical thrombectomy in ischaemic stroke