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Based on the current evidence, we propose that the iron-overloaded, ROS-prone milieu of EMS renders EESCs susceptible to Erastin-triggered, multi-pathway regulated cell death that integrates ferroptosis with necroptotic and pyroptotic signaling, while ferritin handling and mitophagy act as key modulators of susceptibility

An urgent need for rigorous, well-designed clinical trials and mechanistic studies to validate traditional claims, elucidate mechanisms of action, standardize treatments, and integrate these valuable traditional medical systems into evidence-based HACE management exists

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neurological functions Helps maintain nerve cell health and supports proper neurological function